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However, additional studies would be necessary to validate these conclusions

The preliminary results were promising, showing a strong correlation with the standard G6PD enzyme activity assay (75)

NCOA4, nuclear receptor coactivator 4

Oxidative stress increases the generation of ROS and release excess glutamate 48,49

The fact that the excitatory effect of -Glu-Glu on synaptic transmission was blocked by TTX indicates that it most likely resulted from an indirect mechanism involving first the partial effect on NMDAr leading and second the endogenous synaptic release of glutamate and glycine, in turn activating both AMPAr and NMDAr ( The changes in -Glu-Glu in the extracellular medium may thus reflect the production of GSH by astroglial cells